Molecular Mimicry: Infection Inducing Autoimmune Disease
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The reason for this audit is to look at the potential part of atomic mimicry in the pathogenesis of human T-lymphotropic infection sort 1 ((HTLV-1)- related myelopathy/tropical spastic paraparesis (HAM/TSP)). Comp-hensive surveys on the pathogenic systems of HTLV-1-related human ailments are accessible all through the therapeutic writing (Bangham 2000, 2003; Barmak et al. 2003; Jacobson 2002; Levin and Jacobson 1997; Nagai and Osame 2003; Osame 2002).Roughly 25 years prior the ?rst human retrovirus, HTLV-1, was separated (Poeisz et al. 1980).
Consequently, disease with HTLV-1 was appeared to bring about grown-up T-cell leukemia (ATL) and HAM/TSP (Gessain et al. 1985; McFarlin and Blattner 1991; Osame et al. 1986; Poeisz et al. 1980; Yoshida et al. 1987). HTLV-1 may taint up to 30% of individuals in endemic zones and 10-20 million individuals around the world (Barmak et al. 2003; Edlich et al. 2000).
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